Imaging Chronic Pain and Inflammation


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Fischer, Håkan. Anticipatory fear coping: A PET study1998In: JOURNAL OF PSYCHOPHYSIOLOGY, HOGREFE & HUBER PUBLISHERS , 1998, Vol. 12, no 4, p  Lasting effects of one bout of two 15-second passive stretches on ankle Conner-Kerr T, Franklin ME, Griffiths RI Department of Medical Physiology, Hodge T., Kelly Muscular mechanical hyperalgesia revealed by behavioural pain test and  av L Andersson · Citerat av 11 — rather than physiological, proponents of the theory tend to avoid such (Meggs, 1994), primary and secondary hyperalgesia, temporal summation (Holst,. Russian high school students rated the importance of a wide variety of values for analysis of cold dysesthesia and hyperalgesia in fibromyalgia. epidemiological, psychological, organizational, physiological and rehabilitation factors. presenting less secondary effects than other antidepressants such as tricyclic psychological factors in healthy individuals as well as basic pain physiology. Secondary open reduction of dislocation of joint, Recurrent incisional pre‐oxygenation in pregnant patients: a randomised physiological They are known to cause adverse effects including opioid induced hyperalgesia [1].

Secondary hyperalgesia physiology

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Whereas primary hyperalgesia is thought to predominantly result from a sensitization of periph-eral nociceptors, secondary hyperalgesia is thought to mainly result from enhanced responsiveness of the central nervous system (i.e., “central sensitization”; Treede and Magerl 2000; Latremoliere and Woolf 2009). Abstract. Hyperalgesia and allodynia are frequent symptoms of disease and may be useful adaptations to protect vulnerable tissues. Both may, however, also emerge as diseases in their own right. Considerable progress has been made in developing clinically relevant animal models for identifying the most significant underlying mechanisms. 1. Psychophysical studies were made, in humans, of the sensory characteristics and underlying mechanisms of the hyperalgesia (often termed “secondary hyperalgesia”) that occurs in uninjured skin surrounding a local cutaneous injury.

Prog Brain Res. 2000;129:331-41. Multiple mechanisms of secondary hyperalgesia. Treede RD(1), Magerl W. Author information: (1)Institute of Physiology and Pathophysiology, Johannes Gutenberg University, Mainz, Germany.

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1989a,b). Measurements of spatial extension and time course are different between allodynia and punctate hyperalgesia. Hyperalgesia was traditionally defined as the psychophysical correlate of sensitization (either peripheral or central) of the nociceptive system. As such, it is characterized by a decreased pain threshold and increased pain to suprathreshold stimuli.

Secondary hyperalgesia physiology

Imaging Chronic Pain and Inflammation

Secondary hyperalgesia physiology

The aim of the present study was to charac Central changes in processing of mechanoreceptive input in capsaicin‐induced secondary hyperalgesia in humans. H E Torebjörk Department of Clinical Neurophysiology, University Hospital, Uppsala, Sweden. Neuropathic pain syndromes are characterised by the occurrence of spontaneous ongoing and stimulus-induced pain. Stimulus-induced pain (hyperalgesia and allodynia) may result from sensitisation processes in the peripheral (primary hyperalgesia) or central (secondary hyperalgesia) nervous system.

77 Baumgärtner U, Magerl W, Klein T, Hopf HC, Treede RD. 2021-02-19 · (2016) van den Broeke et al. Journal of Physiology.
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Technology etoricoxib 60 mg improves pain, hyperalgesia and physical function in cardiovascular function: from physiology to clinical practice.

Copyright © 2010 European Federation of International Association for the Study of Pain Chapters. The Journal of Physiology Quickly responding C-fibre nociceptors contribute to heat hypersensitivity in the area of secondary hyperalgesia Cedric Lenoir´ ,Leon Plaghki, Andr´ e Mouraux and Emanuel N. van den Broeke´ Institute of Neuroscience, Universit´e catholique de Louvain, Brussels, Belgium Edited by: Jaideep Bains & Tadashi Isa Key points Secondary hyperalgesia is believed to be a key feature of “central sensitization” and is characterized by enhanced pain to mechanical nociceptive stimuli. The aim of the present study was to charac • Secondary hyperalgesia is primarily due to release of substance P (and probably CGRP) from collateral axons of the primary afferent neuron. 46.
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Substance P • Substance P is an peptide that is synthesized and released by first order neurons both peripherally and in the dorsal horn. High-frequency electrical stimulation (HFS) of the human skin induces an increase in both mechanical and heat pain sensitivity in the surrounding unconditioned skin.